Stay up to date with notifications from The Independent

Notifications can be managed in browser preferences.

Eating junk food can damage kidneys as much as diabetes, study finds

Both type 2 diabetes and a high-fat diet can lead to elevated blood sugar levels – and have troubling knock-on effects for people's health

Andrew Griffin
Monday 09 May 2016 17:51 BST
Comments
The mice's junk food diet consisted of consisting of cheese, chocolate bars and marshmallows
The mice's junk food diet consisted of consisting of cheese, chocolate bars and marshmallows (Getty)

Your support helps us to tell the story

From reproductive rights to climate change to Big Tech, The Independent is on the ground when the story is developing. Whether it's investigating the financials of Elon Musk's pro-Trump PAC or producing our latest documentary, 'The A Word', which shines a light on the American women fighting for reproductive rights, we know how important it is to parse out the facts from the messaging.

At such a critical moment in US history, we need reporters on the ground. Your donation allows us to keep sending journalists to speak to both sides of the story.

The Independent is trusted by Americans across the entire political spectrum. And unlike many other quality news outlets, we choose not to lock Americans out of our reporting and analysis with paywalls. We believe quality journalism should be available to everyone, paid for by those who can afford it.

Your support makes all the difference.

Eating a junk food diet can be as damaging to the kidney as diabetes, according to a new study.

The problems caused by eating junk food or a diet high in fat are similar to those found in type 2 diabetes, the new research shows.

The study took rats and fed them a diet of either junk food – consisting of cheese, chocolate bars and marshmallows - for eight weeks or a special food that was high in fat for five weeks.

The researchers then looked at the changes those diets made to the animals’ blood sugar levels and the glucose transports that are in the kidneys. Those transporters have a central role in diabetes and problems with them can lead to significant problems for internal organs.

The study found that the rats with type 2 diabetes had more of certain kinds of glucose transporters and regulatory proteins. But the diet caused similar changes in those same receptors – meaning that it could lead to the same problems as experienced by people with diabetes.

Type 2 diabetes occurs when the body has a problem producing enough insulin or doesn’t react to it properly. When that happens, levels of blood sugar increase, which has knock on effects for organs including the kidneys.

But since those same problems can be found with junk food and high fat diets, similar problems might be seen in the future, the researchers said.

Obesity is biggest threat

"The Western diet contains more and more processed junk food and fat, and there is a well-established link between excessive consumption of this type of food and recent increases in the prevalence of obesity and type 2 diabetes,” said Dr Havovi Chichger, a senior lecturer in biomedical science at Anglia Ruskin University. “In our study, type 1 and type 2 diabetes both induce changes in glucose transport in the kidney, but junk food or a diet high in fat causes changes that are very similar to those found in type 2 diabetes.

The study and those like it may help treat the effects of such problems with high bloody sugar, the researchers said.

"A new treatment for diabetic patients constitutes blocking the glucose transporter in the kidney to reduce blood glucose levels,” Dr Chichger, who was the lead author on the study, said. “Understanding how diet can affect sugar handling in the kidneys and whether the inhibitors can reverse these changes could help to protect the kidneys from further damage."

Join our commenting forum

Join thought-provoking conversations, follow other Independent readers and see their replies

Comments

Thank you for registering

Please refresh the page or navigate to another page on the site to be automatically logged inPlease refresh your browser to be logged in